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antiviral immunity

  • White Spot Syndrome Virus Benefits from Endosomal Trafficking, Substantially Facilitated by a Valosin-Containing Protein, To Escape Autophagic Elimination and Propagate in the Crustacean <span class="named-content genus-species" id="named-content-1">Cherax quadricarinatus</span>
    Virus-Cell Interactions
    White Spot Syndrome Virus Benefits from Endosomal Trafficking, Substantially Facilitated by a Valosin-Containing Protein, To Escape Autophagic Elimination and Propagate in the Crustacean Cherax quadricarinatus

    White spot disease is currently the most devastating viral disease in farmed crustaceans, such as shrimp and crayfish, and has resulted in a severe ecological problem for both brackish water and freshwater aquaculture areas worldwide. Efficient antiviral control of WSSV disease is still lacking due to our limited knowledge of its pathogenesis. Importantly, research on the WSSV infection mechanism is also quite meaningful for the...

    Chuang Meng, Ling-Ke Liu, Dong-Li Li, Rui-Lin Gao, Wei-Wei Fan, Ke-Jian Wang, Han-Ching Wang, Hai-Peng Liu
  • A CD4<sup>+</sup> T Cell-NK Cell Axis of Gammaherpesvirus Control
    Pathogenesis and Immunity
    A CD4+ T Cell-NK Cell Axis of Gammaherpesvirus Control

    Gammaherpesviruses are widespread and cause cancers. CD4+ T cells are a key defense. We found that they defend indirectly, engaging uninfected presenting cells and recruiting innate immune cells to attack infected targets. This segregation of CD4+ T cells from immediate contact with infection helps the immune system to cope with viral evasion. Priming this defense by vaccination offers a way to protect against...

    Clara Lawler, Philip G. Stevenson
  • Long Noncoding RNA Lnc-MxA Inhibits Beta Interferon Transcription by Forming RNA-DNA Triplexes at Its Promoter
    Pathogenesis and Immunity
    Long Noncoding RNA Lnc-MxA Inhibits Beta Interferon Transcription by Forming RNA-DNA Triplexes at Its Promoter

    IAV can be recognized as a nonself molecular pattern by host immune systems and can cause immune responses. However, the intense immune response induced by influenza virus, known as a “cytokine storm,” can also cause widespread tissue damage (X. Z. J. Guo and P. G. Thomas, Semin Immunopathol 39:541–550, 2017, https://doi.org/10.1007/s00281-017-0636-y; S. Yokota, Nihon Rinsho 61:...

    Xinda Li, Guijie Guo, Min Lu, Wenjia Chai, Yucen Li, Xiaomei Tong, Jing Li, Xiaojuan Jia, Wenjun Liu, Dandan Qi, Xin Ye
  • MicroRNA hsa-miR-324-5p Suppresses H5N1 Virus Replication by Targeting the Viral PB1 and Host CUEDC2
    Cellular Response to Infection
    MicroRNA hsa-miR-324-5p Suppresses H5N1 Virus Replication by Targeting the Viral PB1 and Host CUEDC2
    Ashish Kumar, Akhilesh Kumar, Harshad Ingle, Sushil Kumar, Richa Mishra, Mahendra Kumar Verma, Debasis Biswas, Nachimuthu Senthil Kumar, Anamika Mishra, Ashwin Ashok Raut, Akinori Takaoka, Himanshu Kumar
  • Phosphatase Cdc25A Negatively Regulates the Antiviral Immune Response by Inhibiting TBK1 Activity
    Pathogenesis and Immunity
    Phosphatase Cdc25A Negatively Regulates the Antiviral Immune Response by Inhibiting TBK1 Activity
    Dandan Qi, Lei Hu, Tong Jiao, Tinghong Zhang, Xiaomei Tong, Xin Ye
  • Virus-Cell Interactions
    Lnc-ISG20 Inhibits Influenza A Virus Replication by Enhancing ISG20 Expression

    The replication of influenza A virus is regulated by host factors. However, the mechanisms by which lncRNAs regulate IAV infection are not well understood. We identified that lnc-ISG20 is upregulated during IAV infection and is also an interferon-stimulated gene. We demonstrated that lnc-ISG20 can enhance ISG20 expression, which in turn inhibits IAV replication. Our studies indicate that lnc-ISG20 functions as a competing endogenous RNA...

    Wenjia Chai, Jing Li, Qilin Shangguan, Qi Liu, Xinda Li, Dandan Qi, Xiaomei Tong, Wenjun Liu, Xin Ye
  • Cellular Response to Infection
    Defining the Role of Stress Granules in Innate Immune Suppression by the Herpes Simplex Virus 1 Endoribonuclease VHS
    Hannah M. Burgess, Ian Mohr
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