Author's Correction for Pace et al., J. Virol. 80 (18) 9259-9269.
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Journal of Virology, August 2007, p. 8843-8845, Vol. 81, No. 16
0022-538X/07/$08.00+0     doi:10.1128/JVI.02823-07

AUTHOR'S CORRECTION

Population Level Analysis of Human Immunodeficiency Virus Type 1 Hypermutation and Its Relationship with APOBEC3G and vif Genetic Variation

Craig Pace, Jean Keller, David Nolan, Ian James, Silvana Gaudieri, Corey Moore, and Simon Mallal

Centre for Clinical Immunology & Biomedical Statistics, Royal Perth Hospital & Murdoch University, GPO Box X2213, Perth 6847, Australia; Centre for Forensic Science, School of Anatomy & Human Biology, University of Western Australia, Nedlands, Australia; and Department of Clinical Immunology & Biochemical Genetics, Royal Perth Hospital, Wellington St., Perth 6000, Australia

Volume 80, no. 18, p. 9259-9269, 2006. Page 9259, Abstract, line 4: delete "near-full-length."

Page 9259, Materials and Methods, Patient selection, line 3: "(>1,000 nucleotides)" should read "(>400 nucleotides)."

Page 9260, Materials and Methods, Amplification and sequencing of HIV-1 proviral DNA, measurement of pretreatment HIV RNA levels, and HLA and CCR5 genotyping: Add the following as paragraph 2.

The HIV-1 proviral DNA sequences analyzed in this manuscript have been deposited in GenBank with the following sequential accession numbers: EF178299 through EF178434. The study population from which these sequences were derived overlaps substantially (134 of 136) with that described in a previous publication (P. Kiepiela, A. J. Leslie, I. Honeyborne, D. Ramduth, C. Thobakgale, S. Chetty, P. Rathnavalu, C. Moore, K. J. Pfafferott, L. Hilton, P. Zimbwa, S. Moore, T. Allen, C. Brander, M. M. Addo, M. Altfeld, I. James, S. Mallal, M. Bunce, L. D. Barber, J. Szinger, C. Day, P. Klenerman, J. Mullins, B. Korber, H. M. Coovadia, B. D. Walker, and P. J. Goulder, Nature 432:769-775, 2004). For this analysis, we were aware that the design of PCR and sequencing primers has been altered over time to reduce the amplification of hypermutated HIV-1 sequences with large numbers of G-to-A mutations, so analysis was limited to sequences that were obtained prior to the use of these alternative primers. Instances in which sequences in these two data sets are derived from a common source are noted in the relevant GenBank submissions.

We wish to inform the readers that at the time of the submission of this Author's Correction, missing sequences and gaps are represented by the International Union of Pure and Applied Chemistry code "N" in GenBank, which can also denote undetermined bases. Note that sequence gaps are annotated as such for each sequence but that the use of sequence formatting tools will not show annotated gaps. For this reason, the accompanying tableGo includes a summary of the number of bases determined, the number and percentage of nucleotide mixtures, and the number of undetermined bases (i.e., N's) for each accession number. The nine HIV-1 proviral DNA sequences that were excluded from the analysis data are also marked with an asterisk in the tableGo, indicating sequences with insufficient numbers of substitutions relative to the population consensus to provide stable estimates of G->A substitution rates in the analysis of HIV-1 hypermutation.


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Journal of Virology, August 2007, p. 8843-8845, Vol. 81, No. 16
0022-538X/07/$08.00+0     doi:10.1128/JVI.02823-07





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