MDM2-Dependent Inhibition of p53 Is Required for Epstein-Barr Virus B-Cell Growth Transformation and Infected-Cell Survival
J. Virol. Forte and Luftig
83: 2491
Supplemental material
Files in this Data Supplement:
- Supplemental file 1 -
Fig. S1 (Nutlin treatment of BL cells or LCLs with mutant p53 did not induce apoptosis.)
Fig. S2 (Chemical inhibition of IκB kinase β decreased NFκB DNA binding, target gene expression, and induced apoptosis in LCLs.)
Fig. S3 (ICAM1 is an endogenous reporter of NFκB activity in LCLs.)
Fig. S4 (NFκB target genes are not affected by Nutlin treatment in LCLs.
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