Author's Correction for Peters et al., J. Virol. 82 (7) 3500-3508.
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Journal of Virology, July 2008, p. 7248, Vol. 82, No. 14
0022-538X/08/$08.00+0     doi:10.1128/JVI.00954-08

AUTHOR'S CORRECTION

IRF-3 Activation by Sendai Virus Infection Is Required for Cellular Apoptosis and Avoidance of Persistence

Kristi Peters, Saurabh Chattopadhyay, and Ganes C. Sen

Department of Molecular Genetics, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio 44195

Volume 82, no. 7, p. 3500-3508, 2008. Page 3500, column 2, line 8: Reference 13 should be replaced by reference 25a.

Page 3500, column 2, line 10: The first reference 13 should be replaced by reference 25b, and the second reference 13 should be replaced by reference 25c.

Page 3508, References: The following references should be added.

We apologize for the errors and thank D. Kolakofsky for pointing them out to us.

References

    25
  1. Nishio, M., A. Nagata, M. Tsurudome, M. Ito, M. Kawano, H. Komada, and Y. Ito. 2004. Recombinant Sendai viruses with L1618V mutation in their L polymerase protein establish persistent infection, but not temperature sensitivity. Virology 329:289-301.[Medline]
    2. 25
  2. Nishio, M., M. Tsurudome, M. Ito, M. Kawano, H. Komada, and Y. Ito. 2003. Characterization of Sendai virus persistently infected L929 cells and Sendai virus pi strain: recombinant Sendai viruses having Mpi protein shows lower cytotoxicity and are incapable of establishing persistent infection. Virology 314:110-124.[CrossRef][Medline]
    3. 25
  3. Nishio, M., A. Nagata, A. Yamamoto, M. Tsurudome, M. Ito, M. Kawano, H. Komada, and Y. Ito. 2006. The properties of recombinant Sendai virus having the P gene of Sendai virus pi strain derived from BHK cells persistently infected with Sendai virus. Med. Microbiol. Immunol. 195:151-158.[CrossRef][Medline]

Journal of Virology, July 2008, p. 7248, Vol. 82, No. 14
0022-538X/08/$08.00+0     doi:10.1128/JVI.00954-08





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